May 12, 2025, Filed Under: 2019Molecular, Morphological, and Functional Characterization of Corticotropin-Releasing Factor Receptor 1-Expressing Neurons in the Central Nucleus of the Amygdala Citation: Wolfe SA, Sidhu H, Patel RR, Kreifeldt M, D’Ambrosio SR, Contet C, and Roberto M. “Molecular, Morphological, and Functional Characterization of Corticotropin-Releasing Factor Receptor 1-Expressing Neurons in the Central Nucleus of the Amygdala.” eNeuro, 6, 3. Publisher’s Version
May 12, 2025, Filed Under: 2019Intergenerational Effects of Alcohol: A Review of Paternal Preconception Ethanol Exposure Studies and Epigenetic Mechanisms in the Male Germline Citation: Rompala GR and Homanics GE. “Intergenerational Effects of Alcohol: A Review of Paternal Preconception Ethanol Exposure Studies and Epigenetic Mechanisms in the Male Germline.” Alcoholism: Clinical and Experimental Research, 43, 6, Pp. 1032-1045. Publisher’s Version
May 12, 2025, Filed Under: 2019Intergenerational effects of alcohol: a review of paternal preconception ethanol exposure studies and epigenetic mechanisms in the male germline. Citation: GR Rompala and GE Homanics. “Intergenerational effects of alcohol: a review of paternal preconception ethanol exposure studies and epigenetic mechanisms in the male germline..” Alcoholism: Clinical and Experimental Research, Pp. 43:1032-1045. Publisher’s Version Last updated on 11/22/2019
May 12, 2025, Filed Under: 2019Inactivation of a CRF-dependent amygdalofugal pathway reverses addiction-like behaviors in alcohol-dependent rats. Citation: G de Guglielmo, M Kallupi, MB Pomrenze, E Crawford, S Simpson, P Schweitzer, GF Koob, RO Messing, and O. George. “Inactivation of a CRF-dependent amygdalofugal pathway reverses addiction-like behaviors in alcohol-dependent rats..” Nature Communications, 2019, Pp. 1238. Publisher’s Version Last updated on 05/29/2019
May 12, 2025, Filed Under: 2019IL-1β expression is increased and regulates GABA transmission following chronic ethanol in mouse central amygdala. Citation: Patel RR, Khom S, Steinman MQ, Varodayan FP, Kiosses WB, Hedges DM, Vlkolinsky R, Nadav T, Polis I, Bajo M, Roberts AJ, and Roberto M. “IL-1β expression is increased and regulates GABA transmission following chronic ethanol in mouse central amygdala..” Brain, Behavior and Immunity, 75, Pp. 208-219.
May 12, 2025, Filed Under: 2019Increased IL-6 expression in astrocytes is associated with emotionality, alterations in central amygdala GABAergic transmission, and excitability during alcohol withdrawal. Citation: AJ Roberts, S Khom, M Bajo, R Vlkolinsky, I Polis, C Cates-Gatto, M Roberto, and DL Gruol. “Increased IL-6 expression in astrocytes is associated with emotionality, alterations in central amygdala GABAergic transmission, and excitability during alcohol withdrawal..” Brain Behavior and Immunity, Pp. 82:188-202. Publisher’s Version Last updated on 11/22/2019
May 12, 2025, Filed Under: 2019Identification of HIVEP2 as a dopaminergic transcription factor related to substance use disorders in rats and humans Citation: Zhao J, Chen C, Bell RL, Qing H, and Lin Z. “Identification of HIVEP2 as a dopaminergic transcription factor related to substance use disorders in rats and humans.” Translational Psychiatry, 9, 1, Pp. 247. Publisher’s Version Last updated on 05/24/2020
May 12, 2025, Filed Under: 2019Identification of a functional non-coding variant in the GABA (A) receptor α2 subunit of the C57BL/6J mouse reference genome: major implications for neuroscience research. Citation: Mulligan MK, Abreo T, Neuner SM, Parks C, Watkins CE, Houseal MT, Shapaker TM, M Hook, H Tan, Wang X, Ingels J, Peng J, L Lu, CC Kaczorowski, CD Bryant, GE Homanics, and RW Williams. “Identification of a functional non-coding variant in the GABA (A) receptor α2 subunit of the C57BL/6J mouse reference genome: major implications for neuroscience research..” Frontiers in Genetics, 10, Pp. 188. Publisher’s Version Last updated on 05/29/2019
May 12, 2025, Filed Under: 2019The histone deacetylase inhibitorsuberoylanilide hydroxamic acid (SAHA) alleviates depression-like behavior and normalizes epigeneticchanges in the hippocampus during ethanol withdrawal. Citation: WY Chen, H Zhang, E Gatta, EJ Glover, SC Pandey, and AW Lasek. “The histone deacetylase inhibitorsuberoylanilide hydroxamic acid (SAHA) alleviates depression-like behavior and normalizes epigeneticchanges in the hippocampus during ethanol withdrawal..” Alcohol, Pp. 78:79-87. Publisher’s Version Last updated on 11/22/2019
May 12, 2025, Filed Under: 2019The histone deacetylase inhibitor suberoylanilide hydroxamic acid (SAHA) alleviates depression-like behavior and normalizes epigenetic changes in the hippocampus during ethanol withdrawal Citation: Chen WY, Zhang H, Gatta E, Glover EJ, Pandey SC, and Lasek AW. “The histone deacetylase inhibitor suberoylanilide hydroxamic acid (SAHA) alleviates depression-like behavior and normalizes epigenetic changes in the hippocampus during ethanol withdrawal.” Alcohol, 78, Pp. 79-87. Publisher’s Version